The central role of magnesium deficiency in Tourette's syndrome

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Magnesium is the eleventh most abundant element by mass in the human body. The adult body content is 25 g distributed in the skeleton and soft tissues. The chemical is essential in manipulating important biological polyphosphate such as ATP, DNA, and RNA and in functionming enzymes(a).
The central role of magnesium deficiency in Tourette’s syndrome
In the study to hypothesize that magnesium deficiency may be the central precipitating event and common pathway for the subsequent biochemical effects on substance P, kynurenine, NMDA receptors, and vitamin B6 that may result in the symptomatology of Tourette’s syndrome and several reported comorbid conditions, showed that these comorbid conditions and symptomatology include allergy, asthma, autism, attention deficit hyperactivity disorder, obsessive compulsive disorder, coprolalia, copropraxia, anxiety, depression, restless leg syndrome, migraine, self-injurious behavior, autoimmunity, rage, bruxism, seizure, heart arrhythmia, heightened sensitivity to sensory stimuli, and an exaggerated startle response(1).
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Sources
(a) http://en.wikipedia.org/wiki/Magnesium
(1) http://www.ncbi.nlm.nih.gov/pubmed/11863398  http://medicaladvisorjournals.blogspot.com