Patent to Remove Aluminum from Your Brain

www.freepatentsonline.com/20090204058.pdf    `LINK

So how do you think the Aluminum got into your sinus cavity?

Remember it can take up to 3 years to receive a patent for your product.  I know this first hand.  So Johnson and Johnson needs to step up and answer questions as to what caused them to create such a device.

This patent also specifically focuses on the sinus area.  Normally Aluminum is thought to enter the body through Aluminum cans and via your deodorant, not inhalation. 

Comments can be sent to me via “dallasgoldbug  at  gmail  dot  com”

Update.  This is a post I made to medhelp.org.  LINK`http`www.medhelp.org/posts/Migraines–Headaches/Headaches-following-alcohol-consumption-/show/461649`color: rgb(71, 108, 142); text-decoration: underline; `LINK

My intent wasn’t that we need to try this thing out.  It was to shed light on the root cause for the pain.  Aluminum is only thought to enter the body through deodorant and possibly from beverage containers.  The thought of aluminum in your sinus area is only made possible through inhalation.  And I dont know about you but inhaling heavy metals is not something I do on purpose.  The arousal spraying called GeoEngineering by the government /and Chemtrails by those of us who call it like we see it, has several patents for spraying aluminum particulates in the sky to block the harmful rays from the sun (this is the theory so they say)  it also created a green house effect the increases the overall temp (this is so they can attempt to justify the global warming scam and place carbon taxes on everyone.)  They (meaning the scientific community that met at the climate 2010 conference in San Fran outlined the use of millions of tons of aluminum , barium ,and other material to be introduced by way of arousal spray from aircraft.  The only catch is they also are quick to point out they have done no research as to the harmful effects it causes living organisms.  Again not my idea, and I hope you would look it up before screaming conspiracy theorists. 

Anyway, the inhalation of these heavy metals is consistent with what this recent patent discusses.  It also outlines the effects such as reduced blood flow to the frontal area just above the eyes.  So when we drink the reaction of the alcohol cause the inflammation. Here is an excerpt from the Patent: 

BACKGROUND OF THE INVENTION 
In Alzheimer’s Disease (AD), the cleavage of beta amyloid protein precursor from the intracellular membrane often produces a protein AB-42 which is incompletely removed by normal clearance processes. Over time, this protein is deposited as a beta amyloid protein Aβ plaque within brain tissue, leading to the local destruction of neurons. The Aβ plaque deposition is also believed to provoke an inflammatory response by microglia and macrophages. These cells are believed to respond to the plaque deposition by releasing pro-inflammatory cytokines and reactive oxygen species (ROS). Although the inflammatory response may be provoked in an effort to clear the brain tissue of the detrimental plaque, it is now believed that this inflammation also injures local neuronal tissue, thereby exacerbating AD. 

High levels of aluminum, copper, iron, and zinc have been found in the brains of AD patients. For example, Finefrock, J. Am. Geriatr. Soc., 51, 1143-1148, (2003) 

It has been hypothesized by Finefrock, supra, that age-related dyshomeostasis and environmental accumulation are responsible for these high metal levels. 

Furthermore, it is believed that these heavy metals play a critical role in the precipitation of BAP. It is known that BAP binds to these heavy metals and even has highly specific binding sites for copper. Accordingly, high levels of these heavy metals have been found in BAP plaques. Huang, J. Nutrition, 2000, May 130(5S Supp.) 1488S-92S). 

Moreover, since both copper and iron are redox active, these metal-laden deposits act as catalysts for cell-free redox reactions that generate hydrogen peroxide and consequently highly toxic hydroxyl radical. 

Accordingly, it is believed that higher-than-normal levels of heavy metals in the brain are deleterious because they not only promote the deposition of BAP plaques, a portion of them promote oxidative stress when deposited. 

Aluminum is of particular concern. It has long been hypothesized that aluminum plays a critical role in the pathogenesis of AD, although this point has remained controversial. Nonetheless, according to Gupta, Cell Mol. Life Sci., 2005 Jan. 62(2) 143-58, the neurotoxic effects of aluminum are beyond any doubt. Moreover, it has further been reported that, once it has entered the brain, aluminum is fairly persistent. Yokel, Toxicol. Sciences, 64, 77-82 (2001) has hypothesized an aluminum half-life in the rat brain of about 150 days, and in the human brain of about 12 years. According to Yokel, supra, repeated aluminum exposure paired with aluminum persistence produces aluminum accumulation. Therefore, the neurotoxicity and the long-half life of aluminum have made it a potential therapeutic target for AD.